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yeah I mean I agree with all the points Julia makes, but I thought the whole piece was very poorly argued, and the specific things I agreed with didn't support the argument that was being made. The screenshot was particularly bad but IMO also succinctly captured what was wrong with the piece

Is the problem in assuming that including more covariates are always better? Or in asserting that observational evidence + theoretical considerations are often the best option? I'm too dull to tell.

Im sure there are many reasons why this is problematic, at least in clinical psychology, it’s a measurement problem for me. Are there others (more consequential) that I am missing?

More covariates do not equal enhanced partial effects. It’s good in theory, but not in practice. Most items or multidimensional variables are polluted with measurement error.

So when you start adding covariates to partial out shared variance, you actually remove meaningful variable, and leave a greater share of ME and small bits of the original construct.

So by adding in as many covariates as possible, whatever variance your primary predictor of interest, is no longer the same construct you had at the start.

At the risk of making an idiot of myself in public: most of this is not wrong, right? I cannot really comment on the statement that there are usually too few covariates in ph, but it is strictly more likely that you can block the paths that need to be blocked if you have access to more variables.

That is, at least you can ignore something that you do have, rather than control for something that you don't have. So, I guess it is always better to have more data (if there would be no financial and storage constraints of course).

In my view there are two points here: 1) It’s about the comparison with RCTs, and RCTs largely rid you of the need to find & control for all confounders. 2) It’s not clear at all whether more covariates = better, since you might as well end up conditioning on a collider or post-treatment variable.

I'd also include 3) "include covariates in the model" is not using "causal methods" as such, even if focusing on confounders. Like yes, only confounders should be included, but causal do-calculus is broader than simply interrogating whether any individual variable is a confounder.

I think you misunderstand my point. You can decide to leave out colliders only if you have them, but you cannot control for something that you do not happen to have.

Yes you can leave out colliders (and instruments) but this "just collecting more covariates so RCT is no longer needed" approach actually makes the analysis harder not easier, as you have to go through the long list of covariates to decide which to include or exclude.

Chao Wang@excel-wang.bsky.social

So yes the more covariates we have, the higher chances we catch all confounders (this part is true); but it also means the higher chances we catch collider and instrument variables... then you need to think carefully which covariates to adjust.

So yes the more covariates we have, the higher chances we catch all confounders (this part is true); but it also means the higher chances we catch collider and instrument variables... then you need to think carefully which covariates to adjust.

I think the paragraph, and the paragraphs surrounding it, are both talking about modelling the likelihood of treatment; it's using 'data' as a short hand for the data within that model, not the number of covariates in the broader dataset that you have access to

Lord knows I took far too many metrics classes to not feel like a troglodyte for asking this but…what specifically is wrong with this? I’m forgetting so much.

Not stupid at all! I don't think these are obvious problems, which is probably how it got through the editing. But I'm surprised a statistician wrote it...

I mean at least senile! Barely more than two years ago I was taking the most advanced undergrad metrics classes my uni had 💀

This reminds me of a conversation I had with the a principal applied scientist at a certain large firm I once worked at. Guy legitimately thought the best way to do causal inference was to throw as many variables in autoML as we could then do SHAP.

If every principal applied scientist thought the way he did and acted on those thoughts, company would collapse. Thanks god they employ a ton of economists.

If the quarterly theme of Asterisk is ever “Reflection” I’m gonna write an essay that reflects on some of the stinkers that have come through into an otherwise fun mag to read.

Hey Johannes This isn't just another post in your feed. It's a family's story, shared with hope that someone will care. Please visit my page and read the pinned post. ❤️

Besides the last statement, none of this seems unreasonable? A richer dataset will make it easier to adjust for confounding, all else equal. And most PH research probably does suffer more from underadjustment than from overfitting.

Go read the article that the metaculus guy wrote about whether prediction markets are good at predicting things and then go and have a drink.

On giving the whole piece a read, that’s def not a great paragraph but it feels more clumsy writing due to the simple writing/communication style they were going for more than fundamental misunderstanding I think? Can’t know for sure but my initial 😬 is slightly pulled back now.